RNA-dependent RNA polymerase | kinase | tyrosine kinase | Anaplastic lymphoma kinase | Janus kinase 1 | Cyclin D1 | AMP-activated protein kinase | protein kinase | Lyn (Src family Kinase) | cyclin A | Creatine kinase | Bruton's tyrosine kinase | Aurora kinase | Aurora B kinase | anaplastic lymphoma kinase | Voltage-dependent calcium channel | Tyrosine kinase | Src family kinase | Spike-timing-dependent plasticity | Mitogen-activated protein kinase kinase | Limited dependent variable | Light-dependent reactions | Leukocyte receptor tyrosine kinase | Kinase | Janus kinase | Fructosamine-3-kinase | Dependent territory | dependent (record label) | ''dependent'' or ''response'' variable | dependent |
SMAR1 (Scaffold/Matrix attachment region-binding protein 1; also known as BANP), a tumour-suppressor MAR-binding protein that down-regulates Cyclin D1 expression by recruiting HDAC1-mSin3A co-repressor complex at Cyclin D1 promoter locus; SMAR1 is the target of prostaglandin A2 (PGA2) induced growth arrest.
A single cyclin A gene has been identified in Drosophila while Xenopus, mice and humans contain two distinct types of cyclin A: A1, the embryonic-specific form, and A2, the somatic form.
CDK5 is required for proper development of the brain and to be activated, CDK5 must associate with CDK5R1 or CDK5R2.
Altered expression of multiple genes is observed with miR-885-5p, including the CDK2 and MCM5 genes encoding cyclin-dependent kinase 2 and mini-chromosome maintenance protein MCM5, and also with several p53 target genes.
p14ARF inhibits mdm2, thus promoting p53, which promotes p21 activation, which then binds and inactivates certain cyclin-CDK complexes, which would otherwise promote transcription of genes that would carry the cell through the P18
p18 (protein) a member of the INK4 family of cyclin dependent kinase inhibitors.
An atypical form of RTT, characterized by infantile spasms or early onset epilepsy, can also be caused by a mutation to the gene encoding cyclin-dependent kinase-like 5 (CDKL5).
In 2001 he was awarded the Nobel Prize in Physiology or Medicine with Leland Hartwell and Sir Paul Nurse for their discoveries regarding cell cycle regulation by cyclin and cyclin-dependent kinases.